FRS3
genyn codio-protien yn y rhywogaeth Homo sapiens
Protein sy'n cael ei godio yn y corff dynol gan y genyn FRS3 yw FRS3 a elwir hefyd yn Fibroblast growth factor receptor substrate 3 (Saesneg). Segment o DNA yw'r genyn, sy'n amgodio ffwythiant arbennig. Mae'r genyn yma wedi ei leoli ar yr edefyn ôl o gromosom dynol 6, band 6p21.1.[2]
FRS3 | |||||||||||||||||
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Dynodwyr | |||||||||||||||||
Cyfenwau | FRS3, FRS2-beta, FRS2B, FRS2beta, SNT-2, SNT2, fibroblast growth factor receptor substrate 3 | ||||||||||||||||
Dynodwyr allanol | OMIM: 607744 HomoloGene: 4845 GeneCards: FRS3 | ||||||||||||||||
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Species | Bod dynol | Llygoden | |||||||||||||||
Entrez |
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Ensembl |
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UniProt |
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RefSeq (mRNA) |
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RefSeq (protein) |
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Lleoliad (UCSC) | n/a | n/a | |||||||||||||||
PubMed search | [1] | n/a | |||||||||||||||
Wicidata | |||||||||||||||||
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Cyfystyron golygu
Yn aml mae gan enynnau lawer o gyfystyron. Mae hyn oherwydd eu bod yn aml yn cael eu darganfod gan nifer o bobl mewn cyd-destunau gwahanol heb wybod mai'r un genynnau oeddyn nhw. Hefyd mae gan wahanol gymunedau gwyddonol safonau gwahanol ar gyfer enwi genynnau. Dyma restr o gyfystyron ar gyfer y genyn FRS3.
- SNT2
- FRS2B
- SNT-2
- FRS2beta
- FRS2-beta
Llyfryddiaeth golygu
- "SNT-2 interacts with ERK2 and negatively regulates ERK2 signaling in response to EGF stimulation. ". Biochem Biophys Res Commun. 2004. PMID 15485655.
- "FRS2 family docking proteins with overlapping roles in activation of MAP kinase have distinct spatial-temporal patterns of expression of their transcripts. ". FEBS Lett. 2004. PMID 15094036.
- "FRS2beta, a potential prognostic gene for non-small cell lung cancer, encodes a feedback inhibitor of EGF receptor family members by ERK binding. ". Oncogene. 2010. PMID 20228838.
- "Unique role of SNT-2/FRS2beta/FRS3 docking/adaptor protein for negative regulation in EGF receptor tyrosine kinase signaling pathways. ". Oncogene. 2006. PMID 16702953.
- "Broadly expressed SNT-like proteins link FGF receptor stimulation to activators of Ras.". Oncogene. 1996. PMID 8761293.